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當(dāng)前位置:首頁  >  產(chǎn)品中心  >  氣體濃度控制  >  動物低氧濃度控制實(shí)驗(yàn)艙  >  OX-100大號動物低氧實(shí)驗(yàn)系統(tǒng)

大號動物低氧實(shí)驗(yàn)系統(tǒng)

簡要描述:塔望科技提供全系列的動物實(shí)驗(yàn)用低/高氧控制產(chǎn)品,包括恒定濃度控制的低氧動物箱、高氧動物箱、可編程的間歇氧濃度控制系統(tǒng)、帶緩沖艙的手套低氧箱等。整套低氧/高氧實(shí)驗(yàn)箱裝置主要由氧氣控制器和動物實(shí)驗(yàn)箱兩部分組成。另可提供多種不同的氣體控制器,滿足不同實(shí)驗(yàn)O2、CO2、NO、CO、O3等氣體濃度控制的需求。

  • 產(chǎn)品型號:OX-100
  • 廠商性質(zhì):生產(chǎn)廠家
  • 更新時(shí)間:2026-01-20
  • 訪  問  量:2077

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產(chǎn)品描述

塔望科技提供全系列的動物實(shí)驗(yàn)用低/高氧控制產(chǎn)品,包括恒定濃度控制的低氧動物箱、高氧動物箱、可編程的間歇氧濃度控制系統(tǒng)、帶緩沖艙的手套低氧箱等。整套低氧/高氧實(shí)驗(yàn)箱裝置主要由氧氣控制器和動物實(shí)驗(yàn)箱兩部分組成。另可提供多種不同的氣體控制器,滿足不同實(shí)驗(yàn)O2、CO2、NO、CO、O3等氣體濃度控制的需求。

Ox-100動物低氧實(shí)驗(yàn)系統(tǒng)可以控制動物實(shí)驗(yàn)箱內(nèi)持續(xù)低氧的環(huán)境,用以制造相關(guān)的低氧實(shí)驗(yàn)?zāi)P?。用戶可自由設(shè)置所需要的濃度和實(shí)驗(yàn)持續(xù)時(shí)間,所有的設(shè)置通過控制主機(jī)觸摸屏完成,人性化設(shè)計(jì),操作簡便。

Ox-100動物低氧實(shí)驗(yàn)系統(tǒng)監(jiān)測指標(biāo)全面,動物低氧艙內(nèi)具有集成化的傳感器模塊,內(nèi)置溫度、濕度、氧氣、二氧化碳傳感器。可以實(shí)時(shí)監(jiān)測動物低氧艙內(nèi)的環(huán)境。系統(tǒng)通過閉環(huán)反饋控制,根據(jù)動物低氧艙內(nèi)的氧濃度實(shí)時(shí)反饋控制,使動物實(shí)驗(yàn)低氧數(shù)據(jù)更準(zhǔn)確,避免了控制型濃度輸出和低氧艙內(nèi)濃度不一致的情況。Ox-100動物低氧實(shí)驗(yàn)系統(tǒng)具有優(yōu)良的控制性能,持續(xù)低氧實(shí)驗(yàn)時(shí),氧濃度的誤差為0.1%

Ox-100動物低氧實(shí)驗(yàn)系統(tǒng)提供不同尺寸的動物低氧箱,默認(rèn)低氧箱可放置1個(gè)大鼠籠(或2個(gè)小鼠籠),同時(shí)提供大號規(guī)格,可容納2個(gè)大鼠籠和4個(gè)大鼠籠。如需其它規(guī)格,可提供定制。

如需高氧實(shí)驗(yàn),請選擇型號Ox-100HE。

產(chǎn)品特點(diǎn)及參數(shù)

1. 為動物低氧實(shí)驗(yàn)?zāi)P偷慕⑻峁┓€(wěn)定的低氧環(huán)境

2. 按照設(shè)定氣體濃度自動配比氣體,維持恒定的氧氣濃度環(huán)境。無需在箱體外混合比例氣體,實(shí)驗(yàn)氧濃度的準(zhǔn)確,節(jié)省氣源

3. 艙體采用全透明PMMA材質(zhì),防止由于光線影響動物生物節(jié)律

4. 7英寸大屏觸摸屏控制,人性化界面,操作簡單

5. 監(jiān)測參數(shù):溫度、濕度、氧氣O2濃度、二氧化碳濃度

6. 控制精度:±0.1%

7. 非色散紅外(NDIR)二氧化碳傳感器,測量范圍:05000ppm

8. 進(jìn)口電化學(xué)氧氣O2濃度檢測器,測量范圍:0-25%vol,線性度好,檢測準(zhǔn)確、使用壽命長。具有溫度補(bǔ)償機(jī)制

9. 溫度檢測:進(jìn)口高精度溫度傳感器

10. 氧氣濃度變化動態(tài)曲線,直觀了解氧氣濃度變化的過程

11. 具有定時(shí)功能,實(shí)驗(yàn)完成,自動恢復(fù)常氧狀態(tài),并伴有聲音提示

12. 氧氣濃度自動校準(zhǔn):通過控制器對傳感器快速校準(zhǔn)

13. *的氣體混合及循環(huán)機(jī)制,保證箱體內(nèi)氣體濃度的均一

14. 高性能電磁閥,性能穩(wěn)定,超長壽命

15. 艙體尺寸有多種選擇,可靈活搭配。也可根據(jù)實(shí)驗(yàn)要求進(jìn)行定制



ProOx-100動物間歇低氧實(shí)驗(yàn)系統(tǒng)多功能控制


可進(jìn)行間歇低氧實(shí)驗(yàn)(CIH)、急性缺氧實(shí)驗(yàn)、慢性缺氧實(shí)驗(yàn)、高氧/低氧交替實(shí)驗(yàn)



大號動物低氧實(shí)驗(yàn)系統(tǒng)


應(yīng)用領(lǐng)域

肺動脈高壓、腎臟疾病研究、腫瘤研究、心血管疾病研究、視網(wǎng)膜病變、運(yùn)動醫(yī)學(xué)研究、OSAHS、腦發(fā)育與神經(jīng)生物學(xué)、干細(xì)胞研究、醫(yī)學(xué)研究等

型號說明


名稱

型號

說明

單位

動物低氧實(shí)驗(yàn)系統(tǒng)

Ox-100

恒定氧控制,低氧

動物氧濃度實(shí)驗(yàn)系統(tǒng)

Ox-100HE

恒定氧控制,低氧/高氧

動物間歇低氧實(shí)驗(yàn)系統(tǒng)

ProOx-100

恒定氧控制/間歇氧控制,低氧

動物間歇氧濃度實(shí)驗(yàn)系統(tǒng)

ProOx-100HE

恒定氧控制/間歇氧控制,低氧/高氧


艙體型號(可選擇不同尺寸的低氧艙)

名稱

型號

說明

單位

動物實(shí)驗(yàn)艙體小號

OxC-S

大鼠籠x1

動物實(shí)驗(yàn)艙體中號

OxC-M

大鼠籠x2

動物實(shí)驗(yàn)艙體大號

OxC-L

大鼠籠x4

動物實(shí)驗(yàn)艙體特大號

OxC-XL

大鼠籠x8

動物實(shí)驗(yàn)艙體-CIH

OxC-CIH

36只小鼠

手套操作箱

Gl-700

700L


*我公司可提供3Q驗(yàn)證,根據(jù)客戶的特殊應(yīng)用、特殊需求提供功能定制服務(wù),也可以提供相關(guān)的實(shí)驗(yàn)服務(wù),詳情請來電問詢

引用文獻(xiàn)

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[2] Wu L W, Chen M, Jiang C Y, et al. Inactivation of AXL in Cardiac Fibroblasts Alleviates Right Ventricular Remodeling in Pulmonary Hypertension[J]. Advanced Science (IF 14.1), 2025: e08995.

[3] Lei R, Gu M, Li J, et al. Lipoic acid/trometamol assembled hydrogel as injectable bandage for hypoxic wound healing at high altitude[J]. Chemical Engineering Journal (IF 13.4), 2024, 489: 151499.

[4] Li Z, Li H, Qiao W, et al. Multi-omics dissection of high TWAS-active endothelial pathogenesis in pulmonary arterial hypertension: bridging single-cell heterogeneity, machine learning-driven biomarkers, and developmental reprogramming[J]. International Journal of Surgery (IF 10.1), 10.1097.

[5] Pei Y, Huang L, Wang T, et al. Bone marrow mesenchymal stem cells loaded into hydrogel/nanofiber composite scaffolds ameliorate ischemic brain injury[J]. Materials Today Advances (IF 10), 2023, 17: 100349.

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[25] Ding Y, Liu W, Zhang X, et al. Bicarbonate-Rich Mineral Water Mitigates Hypoxia-Induced Osteoporosis in Mice via Gut Microbiota and Metabolic Pathway Regulation[J]. Nutrients, 2025, 17(6): 998.

[26] Gu N, Shen Y, He Y, et al. Loss of m6A demethylase ALKBH5 alleviates hypoxia-induced pulmonary arterial hypertension via inhibiting Cyp1a1 mRNA decay[J]. Journal of Molecular and Cellular Cardiology, 2024.

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[29] Chang P, Xu M, Zhu J, et al. Pharmacological Inhibition of Mitochondrial Division Attenuates Simulated High‐Altitude Exposure‐Induced Memory Impairment in Mice: [30] Involvement of Inhibition of Microglia‐Mediated Synapse Elimination[J]. CNS Neuroscience & Therapeutics, 2025, 31(6): e70473.

[30] Liu C, Qu D, Li C, et al. miR‐448‐3p/miR‐1264‐3p Participates in Intermittent Hypoxic Response in Hippocampus by Regulating Fam76b/hnRNPA2B1[J]. CNS Neuroscience & Therapeutics, 2025, 31(2): e70239.

[31] Wu L W, Chen M, Jiang D J, et al. TCF7 enhances pulmonary hypertension by boosting stressed natural killer cells and their interaction with pulmonary arterial smooth muscle cells[J]. Respiratory Research, 2025, 26(1): 202.

[32] Xie L, Wu Q, Huang H, et al. Neuroregulation of histamine of circadian rhythm disorder induced by chronic intermittent hypoxia[J]. European Journal of Pharmacology, 2025: 177662.

[33] Cai S, Li Z, Bai J, et al. Optimized oxygen therapy improves sleep deprivation-induced cardiac dysfunction through gut microbiota[J]. Frontiers in Cellular and Infection Microbiology, 2025, 15: 1522431.

[34] Wang X, Xie Y, Niu Y, et al. CX3CL1/CX3CR1 signal mediates M1-type microglia and accelerates high-altitude-induced forgetting[J]. Frontiers in Cellular Neuroscience, 2023, 17: 1189348.

[35] He Y, Wang Y, Duan H, et al. Pharmacological targeting of ferroptosis in hypoxia-induced pulmonary edema: therapeutic potential of ginsenoside Rg3 through activation of the PI3K/AKT pathway[J]. Frontiers in Pharmacology, 2025, 16: 1644436.

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[38] Ma Q, Ma J, Cui J, et al. Oxygen enrichment protects against intestinal damage and gut microbiota disturbance in rats exposed to acute high-altitude hypoxia[J]. Frontiers in Microbiology, 2023, 14.

[39] Lan J, Lin J, Guo Y, et al. Sequencing and bioinformatics analysis of exosome-derived miRNAs in mouse models of pancreatic injury induced by OSA[J]. Frontiers in Physiology, 2025, 16: 1712442.

[40] Feng X, Li C, Zhang W, et al. Mechanism of retinal angiogenesis induced by HIF-1α and HIF-2α under hyperoxic conditions[J]. Scientific Reports, 2025, 15(1): 36049.

[41] Yao Y, Chen Y, Li Y, et al. TGM2 Enhances Hypobaric Hypoxia-mediated Brain Injury Via Regulating NLRP3/GSDMD Signaling[J]. Neurochemical Research, 2025, 50(6): 1-11.

[42] Yang A, Guo L, Zhang Y, et al. MFN2-mediated mitochondrial fusion facilitates acute hypobaric hypoxia-induced cardiac dysfunction by increasing glucose catabolism and ROS production[J]. Biochimica et Biophysica Acta (BBA)-General Subjects, 2023: 130413.

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[44] Xu A, Huang F, Chen E, et al. Hyperbaric oxygen therapy attenuates heatstroke-induced hippocampal injury by inhibiting microglial pyroptosis[J]. International Journal of Hyperthermia, 2024, 41(1): 2382162.

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